Figure Data and Table Data for Differential exposure and acute health impacts of inhaled solid-fuel emissions from rudimentary and advanced cookstoves in female CD-1 mice.
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To link health benefits derived from advanced CS usage to increasing combustion efficiencies of the cookstove, a wide variety of general health and respiratory system parameters that are often altered acutely following exposure to air pollutants were assessed. The figures and tables reflect the health outcomes. This dataset is associated with the following publication: Gibbs-Flournoy, E., I. Gilmour, M. Higuchi, J. Jetter, I. George, L. Copeland, R. Harrison, V. Moser, and J. Dye. Differential exposure and acute health impacts of inhaled solid-fuel emissions from rudimentary and advanced cookstoves in female CD-1 mice.. ENVIRONMENTAL RESEARCH. Academic Press Incorporated, Orlando, FL, USA, 161: 35-48, (2018).
The Role of Fuel Type and Combustion Phase on the Toxicity of Biomass Smoke Following Inhalation Exposure in Mice
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This dataset includes physico-chemical characteristics of biomass smoke of three different fuels (oak, peat, and eucalyptus) generated from a tube furnace system at two different combustion phases (smoldering and flaming) and also provides comparisons of lung toxicity outcomes after inhalation and aspiration exposures to the biomass smoke. This dataset is associated with the following publication: Kim, Y.H., C. King, Q. Krantz, M.M. Hargrove, I. George, J. McGee, L. Copeland, M. Hays, M. Landis, M. Higuchi, S. Gavett, and M. Gilmour. The role of fuel type and combustion phase on the toxicity of biomass smoke following inhalation exposure in mice. Archives of Toxicology. Springer, New York, NY, USA, 93(6): 1501-1513, (2019).
The Role of Fuel Type and Combustion Phase on the Toxicity of Biomass Smoke Following Inhalation Exposure in Mice
공공데이터포털
This dataset includes physico-chemical characteristics of biomass smoke of three different fuels (oak, peat, and eucalyptus) generated from a tube furnace system at two different combustion phases (smoldering and flaming) and also provides comparisons of lung toxicity outcomes after inhalation and aspiration exposures to the biomass smoke. This dataset is associated with the following publication: Kim, Y.H., C. King, Q. Krantz, M.M. Hargrove, I. George, J. McGee, L. Copeland, M. Hays, M. Landis, M. Higuchi, S. Gavett, and M. Gilmour. The role of fuel type and combustion phase on the toxicity of biomass smoke following inhalation exposure in mice. Archives of Toxicology. Springer, New York, NY, USA, 93(6): 1501-1513, (2019).
Burn pit smoke condensate-mediated toxicity in human airway epithelial cells
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Data include assessment of transepithelial electrical resistance, lactate dehydrogenase, toxicity scores, pro-inflammatory cytokines, and modulation of gene expression in human airway epithelial cells exposed to burn pit smoke condensates. This dataset is associated with the following publication: Ghosh, A., A. Payton, S. Gallant, K. Rogers, T. Mascenik, E. Hickman, C. Love, K. Schichlein, T. Smyth, Y.H. Kim, J. Rager, M. Gilmour, S. Randell, and i. Jspers. Burn pit smoke condensate-mediated toxicity in human airway epithelial cells. CHEMICAL RESEARCH IN TOXICOLOGY. American Chemical Society, Washington, DC, USA, 37(5): 791-803, (2024).
Simulated burn pit smoke condensates cause sustained impact on human airway epithelial cells
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This dataset provides information on the gene regulation by single and repeated exposure to lower dose of burn pit smoke condensates and biological changes at 48 hrs post-exposure depending on different combustion conditions. The findings suggest that exposure to burn pit smoke condensates may impart a lasting adverse impact on human respiratory health, and the sustained effects depend on the waste source material and combustion condition. This dataset is associated with the following publication: Ghosh, A., K. Rogers, S. Gallant, S. Brocke, A. Speen, Y.H. Kim, I. Gilmour, S. Randell, and i. jaspers. Simulated burn pit smoke condensates cause sustained impact on human airway epithelial cell. TOXICOLOGICAL SCIENCES. Society of Toxicology, RESTON, VA, 204(1): 2-8, (2025).
Burn pit smoke condensate-mediated toxicity in human nasal epithelial cells
공공데이터포털
This dataset provides information about how short-term inhalation of burn pit smoke particles, specifically plastic smoke particles, causes significant toxicity in human nasal epithelial cells and how toxicity caused by inhaled emissions from plastic incinerations could contribute to the development of sinonasal disease associated with burn pits and other environmental particulate matter exposures. This dataset is associated with the following publication: Rogers, K., E. WaMaina, A. Barber, S. Masood, C. Love, Y.H. Kim, M. Gilmour, and i. Jaspers. Emissions from plastic incineration induce inflammation, oxidative stress, and impaired bioenergetics in primary human respiratory epithelial cells. TOXICOLOGICAL SCIENCES. Society of Toxicology, RESTON, VA, 199(2): 301-315, (2024).
Dataset: Smoldering and Flaming Biomass Wood Smoke Inhibit Respiratory Responses in Mice
공공데이터포털
The dataset consists of 2 revised files. The excel file shows all of the individual data used in calculation of the tables and figures. Each table and figure has data stored on a separate tab of the file. The zip file consists of 5 GraphPad Prism files which show the statistics and graphs used in the paper. The names of the 5 files indicate which figures and tables are analyzed statistically and the graphs generated from the data. This dataset is associated with the following publication: Hargrove, M., Y.H. Kim, C. King, C. Wood, M. Gilmour, J. Dye, and S. Gavett. Smoldering and Flaming Biomass Wood Smoke Inhibit Respiratory Responses in Mice. INHALATION TOXICOLOGY. Taylor & Francis, Inc., Philadelphia, PA, USA, 31(6): 236-247, (2019).
Dataset: Smoldering and Flaming Biomass Wood Smoke Inhibit Respiratory Responses in Mice
공공데이터포털
The dataset consists of 2 revised files. The excel file shows all of the individual data used in calculation of the tables and figures. Each table and figure has data stored on a separate tab of the file. The zip file consists of 5 GraphPad Prism files which show the statistics and graphs used in the paper. The names of the 5 files indicate which figures and tables are analyzed statistically and the graphs generated from the data. This dataset is associated with the following publication: Hargrove, M., Y.H. Kim, C. King, C. Wood, M. Gilmour, J. Dye, and S. Gavett. Smoldering and Flaming Biomass Wood Smoke Inhibit Respiratory Responses in Mice. INHALATION TOXICOLOGY. Taylor & Francis, Inc., Philadelphia, PA, USA, 31(6): 236-247, (2019).
Comparative Toxicity of Smoldering Versus Flaming Emissions from Various Biomass Fuels
공공데이터포털
This dataset includes physico-chemical characteristics of biomass smoke of five different fuels (red oak, peat, pine needles, pine, and eucalyptus) generated from a tube furnace system at two different combustion phases (smoldering and flaming) and also provides two toxicological outcomes (lung toxicity in mice and mutagenicity in Salmonella) associated with exposures to the biomass smoke PM collected by a cryo-trap system. This dataset is associated with the following publication: Kim, Y.H., S. Warren, T. Krantz, C. King, R. Jaskot, W.T. Preston, B. George, M. Hays, M. Landis, M. Higuchi, D. DeMarini, and I. Gilmour. Mutagenicity and Lung Toxicity of Smoldering Versus Flaming Emissions from Various Biomass Fuels: Implications for Health Effects from Wildland Fires. ENVIRONMENTAL HEALTH PERSPECTIVES. National Institute of Environmental Health Sciences (NIEHS), Research Triangle Park, NC, USA, 126(1): 1, (2018).